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CORTICOSTEROIDS TEND TO PRODUCE HYPERGLYCEMIA LEADING TO LOSS OF DIABETES CONTROL
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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THE EFFECTIVENESS OF DIURETICS IN PATIENTS WITH UNDERLYING RENAL OR CARDIOVASCULAR DISEASE MAY BE DIMINISHED BY CO-ADMINISTRATON OF ASPIRIN DUE TO INHIBITION OF RENAL PROSTAGLANDINS, LEADING TO DECREASED RENAL BLOOD FLOW AND SALT AND FLUID RETENTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION WITH ANTIPLATELET AGENTS, SUCH AS ASPIRIN, MAY BE ASSOCIATED WITH AN INCREASE IN BLEEDING.
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CO-ADMINISTRATION WITH ANTIPLATELET AGENTS, SUCH AS ASPIRIN, MAY BE ASSOCIATED WITH AN INCREASE IN BLEEDING.
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CO-ADMINISTRATION CAN RESULT IN DIMINISHED HYPOTENSIVE AND HYPONATREMIC EFFECTS OF ACE INHIBITORS DUE TO THE EFFECT OF ASPIRIN ON THE RENIN-ANGIOTENSIN CONVERSION PATHWAY
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CO-ADMINISTRATION CAN RESULT IN DIMINISHED HYPOTENSIVE AND HYPONATREMIC EFFECTS OF ACE INHIBITORS DUE TO THE EFFECT OF ASPIRIN ON THE RENIN-ANGIOTENSIN CONVERSION PATHWAY
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CO-ADMINISTRATION CAN RESULT IN DIMINISHED HYPOTENSIVE AND HYPONATREMIC EFFECTS OF ACE INHIBITORS DUE TO THE EFFECT OF ASPIRIN ON THE RENIN-ANGIOTENSIN CONVERSION PATHWAY
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CO-ADMINISTRATION CAN RESULT IN DIMINISHED HYPOTENSIVE AND HYPONATREMIC EFFECTS OF ACE INHIBITORS DUE TO THE EFFECT OF ASPIRIN ON THE RENIN-ANGIOTENSIN CONVERSION PATHWAY
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CO-ADMINISTRATION CAN RESULT IN DIMINISHED HYPOTENSIVE AND HYPONATREMIC EFFECTS OF ACE INHIBITORS DUE TO THE EFFECT OF ASPIRIN ON THE RENIN-ANGIOTENSIN CONVERSION PATHWAY
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CO-ADMINISTRATION CAN RESULT IN DIMINISHED HYPOTENSIVE AND HYPONATREMIC EFFECTS OF ACE INHIBITORS DUE TO THE EFFECT OF ASPIRIN ON THE RENIN-ANGIOTENSIN CONVERSION PATHWAY
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CO-ADMINISTRATION CAN RESULT IN DIMINISHED HYPOTENSIVE AND HYPONATREMIC EFFECTS OF ACE INHIBITORS DUE TO THE EFFECT OF ASPIRIN ON THE RENIN-ANGIOTENSIN CONVERSION PATHWAY
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CO-ADMINISTRATION CAN RESULT IN DIMINISHED HYPOTENSIVE AND HYPONATREMIC EFFECTS OF ACE INHIBITORS DUE TO THE EFFECT OF ASPIRIN ON THE RENIN-ANGIOTENSIN CONVERSION PATHWAY
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PATIENTS ON ANTICOAGULATION THERAPY ARE AT RISK FOR BLEEDING
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SALICYLIC ACID CAN DISPLACE PROTEIN-BOUND PHENYTOIN LEADING TO A DECREASE IN THE TOTAL CONCENTRATION OF PHENYTOIN
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ASPIRIN CAN DISPLACE WARFARIN FROM PROTEIN BINDING SITES, LEADING TO A PROLONGATION OF BOTH THE PROTHROMBIN TIME AND THE BLEEDING TIME
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ASPIRIN CAN DISPLACE WARFARIN FROM PROTEIN BINDING SITES, LEADING TO A PROLONGATION OF BOTH THE PROTHROMBIN TIME AND THE BLEEDING TIME
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ASPIRIN CAN DISPLACE WARFARIN FROM PROTEIN BINDING SITES, LEADING TO A PROLONGATION OF BOTH THE PROTHROMBIN TIME AND THE BLEEDING TIME
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ASPIRIN CAN DISPLACE WARFARIN FROM PROTEIN BINDING SITES, LEADING TO A PROLONGATION OF BOTH THE PROTHROMBIN TIME AND THE BLEEDING TIME
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ASPIRIN CAN DISPLACE WARFARIN FROM PROTEIN BINDING SITES, LEADING TO A PROLONGATION OF BOTH THE PROTHROMBIN TIME AND THE BLEEDING TIME
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ASPIRIN CAN DISPLACE WARFARIN FROM PROTEIN BINDING SITES, LEADING TO A PROLONGATION OF BOTH THE PROTHROMBIN TIME AND THE BLEEDING TIME
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ASPIRIN CAN DISPLACE WARFARIN FROM PROTEIN BINDING SITES, LEADING TO A PROLONGATION OF BOTH THE PROTHROMBIN TIME AND THE BLEEDING TIME
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ANTACIDS MAY INCREASE ITS EXCRETION
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ANTACIDS MAY INCREASE ITS EXCRETION
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ANTACIDS MAY INCREASE ITS EXCRETION
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ANTACIDS MAY INCREASE ITS EXCRETION
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ANTACIDS MAY INCREASE ITS EXCRETION
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ANTACIDS MAY INCREASE ITS EXCRETION
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ANTACIDS MAY INCREASE ITS EXCRETION
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ANTACIDS MAY INCREASE ITS EXCRETION
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ANTACIDS MAY INCREASE ITS EXCRETION
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ANTACIDS MAY INCREASE ITS EXCRETION
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SALICYLIC ACID CAN DISPLACE PROTEIN-BOUND VALPROIC ACID LEADING TO AN INCREASE IN SERUM VALPROIC ACID LEVELS
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SALICYLIC ACID CAN DISPLACE PROTEIN-BOUND VALPROIC ACID LEADING TO AN INCREASE IN SERUM VALPROIC ACID LEVELS
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ASPIRIN CAN INCREASE THE ANTICOAGULANT ACTIVITYOF HEPARIN, INCREASING BLEEDING RISK
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ASPIRIN CAN INCREASE THE ANTICOAGULANT ACTIVITYOF HEPARIN, INCREASING BLEEDING RISK
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ASPIRIN CAN INCREASE THE ANTICOAGULANT ACTIVITYOF HEPARIN, INCREASING BLEEDING RISK
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ASPIRIN CAN INCREASE THE ANTICOAGULANT ACTIVITYOF HEPARIN, INCREASING BLEEDING RISK
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ASPIRIN CAN INCREASE THE ANTICOAGULANT ACTIVITYOF HEPARIN, INCREASING BLEEDING RISK
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ASPIRIN CAN INCREASE THE ANTICOAGULANT ACTIVITYOF HEPARIN, INCREASING BLEEDING RISK
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SALICYLIC ACID CAN INHIBIT RENAL CLEARANCE OF METHOTREXATE, LEADING TO BONE MARROW TOXICITY, ESPECIALLY IN ELDERLY OR RENAL IMPAIRED PT.
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DIPYRIDAMOLE MAY COUNTERACT THE ANTICHOLINESTERASE EFFECT OF CHOLINESTERASE INHIBITORS, THEREBY POTENTIALLY AGGRAVATING MYASTHENIA GRAVIS
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DIPYRIDAMOLE MAY COUNTERACT THE ANTICHOLINESTERASE EFFECT OF CHOLINESTERASE INHIBITORS, THEREBY POTENTIALLY AGGRAVATING MYASTHENIA GRAVIS
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DIPYRIDAMOLE MAY COUNTERACT THE ANTICHOLINESTERASE EFFECT OF CHOLINESTERASE INHIBITORS, THEREBY POTENTIALLY AGGRAVATING MYASTHENIA GRAVIS
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CORTICOSTEROIDS MAY INCREASE EXCRETION OF THE DRUG
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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CO-ADMINISTRATION OF NON-STEROIDAL ANTI-INFLAMMATORY WITH ASPIRIN MAY INCREASE BLEEDING OR LEAD TO DECREASED RENAL FUNCTION
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